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For example, the anticonvulsant drug valproic acid and the chemotherapeutic agent cisplatin may lower circulating selenium concentrations in treated subjects (122, 123).

Also, supplemental sodium selenite was found to reduce the toxicity of the antibiotic nitrofurantoin and the herbicide paraquat in animal studies (124). A three-year randomized controlled trial in 160 patients with documented coronary artery disease (CAD) and low high-density-lipoprotein (HDL) levels found that a combination of simvastatin (Zocor) and niacin increased HDL2 subfraction levels, inhibited the progression of coronary artery stenosis (narrowing), and decreased the frequency of cardiovascular events (125).

However, help alcoholism individual help alcoholism of selenium cannot be established, and other studies have reported that antioxidant vitamins alone could interfere with the action of HDL-raising drugs, including help alcoholism (126).

However, a similar trial in an American cohort with higher baseline plasma selenium drug lab found no effect help alcoholism selenium supplementation on SEPP1 concentrations (128). Eating a varied diet and taking a daily MVM supplement should provide sufficient selenium for most people in the US and help improve selenium status in populations with lower selenium intakes outside the US.

At present, the effect of selenium supplementation on cancer risk is not clear enough to support a general recommendation for an extra selenium supplement, especially in men with serum selenium concentrations consistent with adequate selenium intakes. At present, there is no clinical evidence showing that selenium supplementation above recommended levels decreases the risk of breast cancer although some, help alcoholism not all, observational studies have found an inverse relationship between selenium status and breast cancer in women (59).

Aging has Eskalith (Lithium Carbonate)- FDA been associated with significant changes in the requirement for selenium. Originally written in 2001 by: Jane Higdon, Ph. Linus Pauling Institute Oregon State University Updated in October 2003 by: Jane Higdon, Ph. Linus Pauling Institute Oregon State University Updated in November 2007 by: Victoria J.

Linus Pauling Institute Oregon State University Updated in November 2014 by: Barbara Delage, Ph. Linus Pauling Institute Oregon State University Help alcoholism in June 2015 by: Petra A. Assistant Professor Department of Biological Sciences Towson University 1. The importance of selenium help alcoholism human help alcoholism. Mangiapane E, Pessione A, Pessione E. Selenium and selenoproteins: an overview on different biological systems.

Curr Protein Pept Sci. Mariotti M, Ridge PG, Zhang Y, et al. Composition and evolution of the vertebrate and mammalian selenoproteomes.

Terry EN, Diamond JA. In: Erdman Jr J, Macdonald J, Zeisel S, eds. Present Knowledge in Nutrition. Boitani C, Puglisi R. Selenium, help alcoholism key element in spermatogenesis and male fertility.

Adv Exp Help alcoholism Biol. Lu J, Holmgren A. The thioredoxin antioxidant system. Free Radic Biol Med.

Selenium, selenoproteins bone marrow transplantation journal the thyroid gland: interactions in health and disease.

Hill KE, Wu S, Motley AK, et al. Production of selenoprotein P (Sepp1) by hepatocytes is central to selenium homeostasis. Olson GE, Winfrey VP, Hill KE, Burk RF. Megalin mediates selenoprotein P uptake by kidney proximal tubule epithelial cells. Misu H, Takamura T, Takayama H, et al. A liver-derived secretory protein, selenoprotein P, causes help alcoholism resistance. Jeong D, Kim TS, Chung YW, Lee BJ, Kim IY.

Selenoprotein W is a glutathione-dependent antioxidant in vivo. Reeves MA, Hoffmann PR. Cell Help alcoholism Life Sci. Reszka E, Jablonska E, Gromadzinska J, Wasowicz W. Relevance of selenoprotein transcripts for selenium status in humans. Chung YW, Jeong D, Noh OJ, et al. Antioxidative role of selenoprotein W in oxidant-induced mouse embryonic neuronal cell death. Jeon YH, Park YH, Kwon JH, Lee JH, Help alcoholism IY. Inhibition of help alcoholism binding to Rictor of mTORC2 for Akt phosphorylation at Ser473 is regulated by selenoprotein W.

Jeon YH, Park YH, Lee JH, Hong JH, Kim IY. Selenoprotein W enhances skeletal muscle differentiation by inhibiting TAZ binding to 14-3-3 protein. Alkan Z, Duong FL, Hawkes WC. Selenoprotein W controls epidermal growth factor receptor surface expression, activation and degradation via receptor ubiquitination. Ganichkin OM, Xu XM, Carlson BA, et al.

Structure and catalytic mechanism of eukaryotic selenocysteine oil shark liver Lee BC, Peterfi Z, Hoffmann FW, et al. Kumaraswamy E, Malykh A, Korotkov KV, et al.



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